Projects
Eight funded projects
Dissecting non-canonical from canonical Gαi-dependent pathways in ischemia- reperfusion injury

Cardiovascular diseases and stroke belong to the leading causes of death worldwide. After ischemia, the subsequent reperfusion process promotes a local inflammation mediated by infiltrating neutrophils and ROS, exacerbating tissue damage and leading to reperfusion injury. G protein signal transduction modulates cell recruitment into inflammatory tissue, however Gi-driven canonical vs. non-canonical contribution is still elusive..
Mode of action of the Gαi/ Gpsm2 and the RGS4 / neurabin complex in cochlear hair cells

During the development of the inner ear, Gai3 and its non-canonical regulator Gpsm2 play an essential role for orienting sensory hair cells. In addition, in the mature organ of Corti, Gai3 signalling is involved in protective mechanisms during noise trauma.
Modulation of heterotrimeric G protein activity by nucleoside diphosphate kinase

Nucleoside diphosphate kinases (NDPKs) form complexes with heterotrimeric G proteins and provide GTP for the activation of these signal transducers. Thus, their function as activator of G protein signaling (AGS) is investigated in this project.